MSDS

Bayer Environmental Science
Material Safety Data Sheet
MSDS Number: 102000021162
BAYER ADVANCED SEASON LONG WEED CONTROL
FOR LAWNS CONCENTRATE
MSDS Version 1.0
Page 5 of 8
Assessment Carcinogenicity
2,4-D was not carcinogenic in lifetime feeding studies in rats and mice. EPA has classified 2,4-D as a
Group D chemical [not classifiable as to human carcinogenicity].
MCPP-p was not carcinogenic in a chronic feeding study in rats. In an oncogenicity study in mice, there
was an increased incidence of liver tumors in female mice at high doses. EPA classified MCPP-p as
"suggestive of carcinogenicity, but not sufficient to assess human carcinogenic potential."
Dicamba was not carcinogenic in lifetime feeding studies in rats and mice. EPA has classified dicamba
"not likely to be carcinogenic to humans."
Isoxaben is currently listed as a possible human carcinogen based on evidence of benign liver ademonas
at the high dose only in the carcinogenic study in mice.
ACGIH
None.
NTP
None.
IARC
None.
OSHA
None.
Reproductive toxicity
REPRODUCTION:
2,4-D caused reproductive toxicity at dose levels above the saturation of renal
clearance in multi-generation studies in rats.
MCPP-p was not a reproductive toxicant in a two-generation study in rats.
Dicamba was not a primary reproductive toxicant in a two-generation
reproduction study in rats. Reproductive effects were observed only at doses
that caused parental systemic toxicity. Offspring toxicity was manifested at a
dose lower than parental systemic toxicity.
Isoxaben was not a reproductive toxicant in a multi-generation study in rats.
DEVELOPMENTAL TOXICITY:
2,4-D is not a primary developmental toxicant in rats and rabbits. Developmental
effects were observed at doses that caused maternal toxicity.
MCPP-p was not a primary developmental toxicant in rats and rabbits.
Developmental effects were observed in rats but were considered secondary to
maternal toxicity.
Dicamba was not a primary developmental toxicant in rats and rabbits.
Developmental effects were observed in rabbits but were considered secondary
to maternal toxicity.