SDS

Road Rescue Asphalt: SDS for Limestone Rock Asphalt (LRA)
March 2015
SECTION XI – TOXICOLOGICAL INFORMATION, CONTD.
Carbon Dioxide:
Exposure route: Inhalation.
Target organs: Respiratory system, cardiovascular system.
Acute effect: Inhalation of carbon dioxide may cause dizziness, headache, and elevated blood pressure. Inhalation of high
concentrations of this gas may cause hyperventilation and unconsciousness.
Chronic effect/carcinogenicity: Information on chronic effect of prolonged exposure to this substance is not documented.
Hydrogen Sulfide:
Exposure route: Inhalation, skin/eye contact
Target organs: Eyes, respiratory system, central nervous system.
Acute effect: Inhalation, even at small levels can cause fatigue, headache, apnea, lung edema, coma, insomnia, irritability of the
eyes and respiratory system, dizziness and central nervous system effects. In some cases respiratory paralysis leading to death can
occur.
Chronic effect/carcinogenicity: Prolonged exposure to this material can cause toxicosis in people exposed to large concentrations.
Not classifiable as a human carcinogen.
Under certain circumstances, Hydrogen sulfide, a highly flammable and toxic gas, may be released from molten asphalt. H
2
S is a
colorless gas with an odor similar to rotten eggs. Odor cannot be relied on as a means of detection because the olfactory nerves
(sense of smell) rapidly become insensitive to it. In addition, the H
2
S odor may be masked by the general odor of hot asphalt.
Low concentrations (50 – 100 ppm) of H
2
S can irritate the eyes and respiratory tract, and may cause nervousness, cough, nausea,
and headache. Prolonged exposure to concentrations between 250 – 600 ppm, may cause pulmonary edema (fluid in the lungs)
and bronchial pneumonia. Brief exposure to concentrations above 500 ppm can cause unconsciousness and may be fatal. The
OSHA PEL is 20 ppm (ceiling). The ACGIH TLV is 10 ppm with a STEL of 15 ppm. H
2
S may accumulate in an enclosed space.
Persons should stand upwind and avoid breathing the gas when opening hatches and dome covers.
Nitrogen Dioxide:
Exposure route: Inhalation, ingestion, skin/eye contact
Target organs: Eyes, skin, cardiovascular system and respiratory system.
Acute effect: Inhalation can cause burning of the respiratory tract, sore throat, cough, lung edema, dizziness, headache, apnea,
weakness and vomiting. Contact with the skin and/or eyes will cause redness, pain and possibly severe burns. Exposure to very
high concentrations may lead to death.
Chronic effect/carcinogenicity: Not classifiable as a human carcinogen. Prolonged exposure to this material may cause increased
susceptibility to respiratory infection may aggravate asthma and allergic disorders.
Ozone:
Exposure routes: Inhalation, skin/eye contact.
Target organs: Eyes, skin and respiratory system.
Acute effect: Inhalation may cause irritation of the respiratory tract, cough, headache, shortness of breath, asthmatic reactions and
sore throat. Contact with the eyes will result in irritation, pain and redness and may result in loss of vision. It may cause effects
on the central nervous system resulting in headache and impaired vigilance and performance.
Chronic effect/carcinogenicity: Not classifiable as a human carcinogen.